Research study: "*Localization of stuttering based on causal brain lesions*" (2024, 27 May) *Goal:* Here, we investigated the neuroanatomical substrate of stuttering using three independent datasets: (i) case reports from the published literature of acquired neurogenic stuttering following stroke (n = 20, 14 males/six females, 16–77 years); (ii) a clinical single study cohort with acquired neurogenic stuttering following stroke (n = 20, 13 males/seven females, 45–87 years); and (iii) adults with persistent developmental stuttering (n = 20, 14 males/six females, 18–43 years). We used the first two datasets and lesion network mapping to test whether lesions causing acquired stuttering map to a common brain network. We then used the third dataset to test whether this lesion-based network was relevant to developmental stuttering. *Findings:* Findings overlapped in the left-sided posteroventral putamen (which is responsible for automation and internal timing for sequencing of complex motor movements of the lips and other articulators), including the ventral claustrum and amygdalostriatal transition area. Of the many theoretical accounts of stuttering proposed previously, our data provide support for a crucial role of the basal ganglia.