Dysregulation in the Dopaminergic System and Stuttering: My Theory
Content
Dysregulation in the Dopaminergic System and Stuttering: My Theory Dysregulation in the dopaminergic system could be a cause of stuttering. I don't mean to say that we have an excess of dopamine or vice versa, but rather the entire system is dysregulated in ways that we don't understand yet. This dysregulation could be caused by more static factors such as brain damage, but I suspect that most stutterers are impacted by a dynamic issue that we could possibly have treatments for that we haven't discovered yet. I will provide a variety of explanations as to the unique characteristics of stuttering and how it could match up with this theory. Stuttering could vary wildly from person to person because this dysregulation could be more pronounced in some stutterers compared to other stutterers. Furthermore, facial tics are linked to abnormalities in the dopaminergic system. My primary hypothesis as to why facial tics accompany stuttering is that facial tics could be caused by 'haywire' neurons spurred on by the existence of the abnormal dopaminergic system that, for some reason, is at first initiated by simply speaking. In metaphorical terms: Speech is the trigger, and facial tics are the bullet. I honestly wouldn't be opposed to categorizing facial tics as a localized mini-seizure if this is the cause. This could also explain why stuttering can spontaneously appear with the use of drugs that are known to cause changes in the dopaminergic system. Drugs that alter the dopaminergic pathways and sometimes cause stuttering as a side effect are as follows: Methylphenidate, Aripiprazole, Phenothiazine, and most certainly others! In other words, stuttering not to dissimilar from what you experience can be caused by drugs that could cause dysregulation in the dopaminergic system in people who would otherwise have perfectly fluent speech. Isn't that convenient? Caffeine also impacts the dopaminergic system, and although this effect is generally characterized as positive, I strongly suspect that caffeine could negatively impact people with pre-existing dysregulation of the dopaminergic system. This could be why you stutter more with caffeine, but it could also be the reason as to why a select few stutterers stutter less with caffeine. Anything that impacts the dopaminergic system could impact your stutter in both positive and negative ways. I don't entirely understand why that is, but from what I can tell, this seems to be the case. I decided to research stuttering on my own and this is the best theory that I could come up with with the tools that I have. I also apologize if this post seems rather rushed, as I didn't spend a crazy amount of time on this post, so it may not flow as well as it could. I ultimately hope this posts makes people think about stuttering as a dynamic issue rather than a static issue that can't be changed.