I’m a person who stutters and speech-language pathologist with experience working with the brain injury population. I’m not too well-versed with acquired neurogenic stuttering or Neuralink. However, I wouldn’t rule anything as possible future treatments given how little we know at the moment. We need to have enough scientific evidence through randomized control trials with this particular population before we can definitively say it’s effective. Also, those with lesions to Broca’s area typically have apraxia of speech rather than stuttering. However, studies show that those with developmental stuttering have less white matter density in the arcuate fasciculus (the tract connecting Wernicke’s and Broca’s areas). Given the proximity of Broca’s area and the arcuate fasciculus, it wouldn’t be surprising if damage to one or both regions manifested as stuttering or stuttering-like characteristics. Based on what we know so far, behavioral and compensatory approaches have the most evidence for communication impairments resulting from brain injury. It requires frequent practice and repetition to promote neuroplasticity, especially in the first 6-12 months post-injury. For neurogenic stuttering, this may be working on fluency-shaping techniques or using other modes of communication to supplement speech. Given how unique every brain is, each person responds differently to different treatments so there’s no one-size-fits-all approach.