commentr/StutterNovember 8, 2023

Content

I've read scientific articles in the high hundreds, and only a portion of those in direct reference to stuttering. Scientific literature on stuttering is a bit cynical in that it's too zoomed in neural phenomena which it addresses through a lens of structural deficiency. "Speech requires the coordination of hundreds of muscles, and so it is a complicated issue because the brain is complicated and we do not understand the brain. As such, we will point out here how the brain of a stutterer differs from the brain of a fluent person, and deem it a structural permanent issue." I do not, nor will ever accept that proposition. It's plain ignorant. I may accept I will not be the one to derive the answer. Is my brain suddenly changing structure when I'm alone, or with people I feel comfortable with, or talking to babies or pets? Differences between white matter tracts between stutterers and fluents? Okay, but research shows meditation nurtures white matter formation in a matter of weeks. Reduced Default Mode Network (DMN) connectivity but higher level of activity compared to fluents? Okay, we know the DMN is associated to resting states and becomes inhibited during motor execution. We know the DMN is associated to self-referential thoughts. Instead of copping out with structural issue, how about we turn our focus to behavioral differences in resting states? Start with inner speech. Let's also observe RTN/pFRG and Botzinger Complex neuron respiratory patterns during prolonged resting states. Dopaminergic excess in the basal ganglia with multiple competing signals? Impaired connection between the ventral premotor cortex and basal ganglia? Okay, we know the VPMC projects to the striatum. We know the hyperdirect dopamine pathway bypasses the striatum and projects to the subthalamic nucleus. The hyperdirect pathway suppresses or inhibits movement. There's behavioral input here that's contributing to this dynamic. Yet, impairment of this, impairment of that, but no causal connection through an integrative approach. Our brainwaves elicit alpha frequencies of 8-12 Hz when we close our eyes or in resting states, contributing to the continuity of our perception. The hyperdirect pathway interrupts (freezes) our visual field. The trigeminal nerve is a nerve from which 3 branches originate: Ophthalmic nerve (which innervates structures of the eye), maxillary nerve, and mandibular nerve. The eyes, the upper jaw, the lower jaw, are innervated from one source. Why not explore this in connection to stuttering? There's a big link between visual modulation and speech, I just can't put it in words. Ironically enough, none of my findings have directly contributed to improvement of my stutter. I've been through the gutter and tested my spirit in more ways than I can say. No one can put me through worse hell than I put myself in. This pursuit has hardened me, and I simply lost my patience to care about other people's opinions, especially to fear going into sales due to their opinions or whatever. Funnily enough, everyone loves me despite of my stutter. I can't teach people this. I have compiled a few theories and hypotheses, but sharing is futile since I didn't them to directly help me. I'm a salesman from a third-world country who sells paints and construction materials. I have no formal qualifications in neuroscience, medicine, biology, chemistry or whatever. I have the balls to be delusional and blindly pursue a cure with everything stacked against me. I may be giving up on this, but I respect myself for that. The fancy scientists in their comfort zones, no.

Themes

Causes & VariabilityIdentity & Disability

Subthemes

Neurological & BrainTrauma & PsychologicalIdentity & Self-Perception